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How to find us Y. (Yana) Geng, MSc


How does hepatic lipid accumulation lead to lipotoxicity in non-alcoholic fatty liver disease?

Role of Oxidative Stress in the Pathogenesis of Non-Alcoholic Fatty Liver Disease: Implications for Prevention and Therapy

Chemical hypoxia induces pro-inflammatory signals in fat-laden hepatocytes and contributes to cellular crosstalk with Kupffer cells through extracellular vesicles

Extracellular vesicles derived from fat-laden hepatocytes undergoing chemical hypoxia promote a pro-fibrotic phenotype in hepatic stellate cells

Hesperetin protects against palmitate-induced cellular toxicity via induction of GRP78 in hepatocytes

Lipotoxicity in Non-alcoholic Fatty Liver Disease: Mechanisms and Prevention in Experimental Models

Metformin protects against diclofenac - induced toxicity in primary rat hepatocytes by preserving mitochondrial integrity via an alternative pathway involving epac

Protective effect of metformin against palmitate-induced hepatic cell death

Chemical Hypoxia Determines Extracellular Vesicles Release from Fat-Laden Hepatocytes and Cellular Crosstalk with Hepatic Stellate Cells

Protective Effect of Metformin Against Palmitic Acid-Induced Hepatic Cell Death

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