Skip to ContentSkip to Navigation
How to find us drs. K.E.C. (Kaj) Blokland


Regulation of cellular senescence by extracellular matrix during chronic fibrotic diseases

Self DNA perpetuates IPF lung fibroblast senescence in a cGAS-dependent manner

Senescence of IPF Lung Fibroblasts Disrupt Alveolar Epithelial Cell Proliferation and Promote Migration in Wound Healing

Extracellular vesicles in lung health, disease, and therapy

STAT3 Regulates the Onset of Oxidant-induced Senescence in Lung Fibroblasts

Sustained and regulated gene expression by Tet-inducible "all-in-one" retroviral vectors containing the HNRPA2B1-CBX3 UCOE®

Fibroblast senescence in the pathology of idiopathic pulmonary fibrosis

Mitochondrial dysfunction contributes to the senescent phenotype of IPF lung fibroblasts

Read more