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Homologous recombination-deficient cancers: approaches to improve treatment and patient selection

BRCA2 deficiency instigates cGAS-mediated inflammatory signaling and confers sensitivity to tumor necrosis factor-alpha-mediated cytotoxicity

Inflammatory signaling in genomically instable cancers

Progression through mitosis promotes PARP inhibitor-induced cytotoxicity in homologous recombination-deficient cancer cells

Therapeutic targeting and patient selection for cancers with homologous recombination defects

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