Publication

Pathogenesis of vasculitis

Kallenberg, C. & Heeringa, P., 1998, In : Lupus. 7, 4, p. 280-284 5 p.

Research output: Contribution to journalReview articleAcademicpeer-review

APA

Kallenberg, C., & Heeringa, P. (1998). Pathogenesis of vasculitis. Lupus, 7(4), 280-284.

Author

Kallenberg, Cees ; Heeringa, Peter. / Pathogenesis of vasculitis. In: Lupus. 1998 ; Vol. 7, No. 4. pp. 280-284.

Harvard

Kallenberg, C & Heeringa, P 1998, 'Pathogenesis of vasculitis', Lupus, vol. 7, no. 4, pp. 280-284.

Standard

Pathogenesis of vasculitis. / Kallenberg, Cees; Heeringa, Peter.

In: Lupus, Vol. 7, No. 4, 1998, p. 280-284.

Research output: Contribution to journalReview articleAcademicpeer-review

Vancouver

Kallenberg C, Heeringa P. Pathogenesis of vasculitis. Lupus. 1998;7(4):280-284.


BibTeX

@article{c04c11e681f8405eb16dd4078a7aff04,
title = "Pathogenesis of vasculitis",
abstract = "This review discusses current thoughts on the pathogenesis of vasculitis. Secondary vasculitides, frequently associated with infections or systemic autoimmune diseases, are, in most cases, characterized by immune deposits in the vessel wall, which probably underlies the development of lesions. In the primary vasculitides, immune deposits are generally absent. A group of primary vasculitides is, however, strongly associated with anti-neutrophil cytoplasmic autoantibodies (ANCA). Various in vitro and in vivo experimental data suggest that those ANCA are involved in the pathogenesis of lesions in the associated disorders.",
keywords = "systemic vasculitis, anti-neutrophil cytoplasmic antibodies, Wegener's granulomatosis, pathophysiology, HUMAN ENDOTHELIAL-CELLS, ANTINEUTROPHIL CYTOPLASMIC AUTOANTIBODIES, WEGENERS-GRANULOMATOSIS, SYSTEMIC VASCULITIS, ANTIMYELOPEROXIDASE ANTIBODIES, EXPRESS PROTEINASE-3, NEUTROPHILS, GLOMERULONEPHRITIS, MYELOPEROXIDASE, ANCA",
author = "Cees Kallenberg and Peter Heeringa",
note = "Review 6",
year = "1998",
language = "English",
volume = "7",
pages = "280--284",
journal = "Lupus",
issn = "0961-2033",
publisher = "SAGE Publications Inc.",
number = "4",

}

RIS

TY - JOUR

T1 - Pathogenesis of vasculitis

AU - Kallenberg, Cees

AU - Heeringa, Peter

N1 - Review 6

PY - 1998

Y1 - 1998

N2 - This review discusses current thoughts on the pathogenesis of vasculitis. Secondary vasculitides, frequently associated with infections or systemic autoimmune diseases, are, in most cases, characterized by immune deposits in the vessel wall, which probably underlies the development of lesions. In the primary vasculitides, immune deposits are generally absent. A group of primary vasculitides is, however, strongly associated with anti-neutrophil cytoplasmic autoantibodies (ANCA). Various in vitro and in vivo experimental data suggest that those ANCA are involved in the pathogenesis of lesions in the associated disorders.

AB - This review discusses current thoughts on the pathogenesis of vasculitis. Secondary vasculitides, frequently associated with infections or systemic autoimmune diseases, are, in most cases, characterized by immune deposits in the vessel wall, which probably underlies the development of lesions. In the primary vasculitides, immune deposits are generally absent. A group of primary vasculitides is, however, strongly associated with anti-neutrophil cytoplasmic autoantibodies (ANCA). Various in vitro and in vivo experimental data suggest that those ANCA are involved in the pathogenesis of lesions in the associated disorders.

KW - systemic vasculitis

KW - anti-neutrophil cytoplasmic antibodies

KW - Wegener's granulomatosis

KW - pathophysiology

KW - HUMAN ENDOTHELIAL-CELLS

KW - ANTINEUTROPHIL CYTOPLASMIC AUTOANTIBODIES

KW - WEGENERS-GRANULOMATOSIS

KW - SYSTEMIC VASCULITIS

KW - ANTIMYELOPEROXIDASE ANTIBODIES

KW - EXPRESS PROTEINASE-3

KW - NEUTROPHILS

KW - GLOMERULONEPHRITIS

KW - MYELOPEROXIDASE

KW - ANCA

M3 - Review article

VL - 7

SP - 280

EP - 284

JO - Lupus

JF - Lupus

SN - 0961-2033

IS - 4

ER -

ID: 2010810