Publication

KRS: A cut away from release in exosomes

Rabouille, C., 3-Jul-2017, In : The Journal of Cell Biology. 216, 7, p. 1891-1893 3 p.

Research output: Contribution to journalEditorialAcademicpeer-review

APA

Rabouille, C. (2017). KRS: A cut away from release in exosomes. The Journal of Cell Biology, 216(7), 1891-1893. https://doi.org/10.1083/jcb.201706039

Author

Rabouille, Catherine. / KRS : A cut away from release in exosomes. In: The Journal of Cell Biology. 2017 ; Vol. 216, No. 7. pp. 1891-1893.

Harvard

Rabouille, C 2017, 'KRS: A cut away from release in exosomes', The Journal of Cell Biology, vol. 216, no. 7, pp. 1891-1893. https://doi.org/10.1083/jcb.201706039

Standard

KRS : A cut away from release in exosomes. / Rabouille, Catherine.

In: The Journal of Cell Biology, Vol. 216, No. 7, 03.07.2017, p. 1891-1893.

Research output: Contribution to journalEditorialAcademicpeer-review

Vancouver

Rabouille C. KRS: A cut away from release in exosomes. The Journal of Cell Biology. 2017 Jul 3;216(7):1891-1893. https://doi.org/10.1083/jcb.201706039


BibTeX

@article{6fe395c9c9b84e5aaf983bfb5dfc4029,
title = "KRS: A cut away from release in exosomes",
abstract = "Cancer cells often trigger an inflammatory process, which in some cases may be driven by the presence of lysyltRNA synthetase (KRS) in the medium. Kim et al. (2017. J. Cell Biol. https://doi.org/10.1083/jcb.201605118) now demonstrate that cleavage of the KRS by caspase-8 inside cells triggers its interaction with syntenin and its release in inflammatory exosomes.",
keywords = "SECRETION, BIOGENESIS, ALIX",
author = "Catherine Rabouille",
year = "2017",
month = jul,
day = "3",
doi = "10.1083/jcb.201706039",
language = "English",
volume = "216",
pages = "1891--1893",
journal = "The Journal of Cell Biology",
issn = "0021-9525",
publisher = "ROCKEFELLER UNIV PRESS",
number = "7",

}

RIS

TY - JOUR

T1 - KRS

T2 - A cut away from release in exosomes

AU - Rabouille, Catherine

PY - 2017/7/3

Y1 - 2017/7/3

N2 - Cancer cells often trigger an inflammatory process, which in some cases may be driven by the presence of lysyltRNA synthetase (KRS) in the medium. Kim et al. (2017. J. Cell Biol. https://doi.org/10.1083/jcb.201605118) now demonstrate that cleavage of the KRS by caspase-8 inside cells triggers its interaction with syntenin and its release in inflammatory exosomes.

AB - Cancer cells often trigger an inflammatory process, which in some cases may be driven by the presence of lysyltRNA synthetase (KRS) in the medium. Kim et al. (2017. J. Cell Biol. https://doi.org/10.1083/jcb.201605118) now demonstrate that cleavage of the KRS by caspase-8 inside cells triggers its interaction with syntenin and its release in inflammatory exosomes.

KW - SECRETION

KW - BIOGENESIS

KW - ALIX

U2 - 10.1083/jcb.201706039

DO - 10.1083/jcb.201706039

M3 - Editorial

VL - 216

SP - 1891

EP - 1893

JO - The Journal of Cell Biology

JF - The Journal of Cell Biology

SN - 0021-9525

IS - 7

ER -

ID: 48317862